SenseMe — The Science · 219 signals & markers, every one traceable

DATA ARCHITECTURE

Built on 130+ years of clinical science.

SenseMe doesn't invent the science. We integrate it — combining 7 validated clinical instruments cited from peer-reviewed journals, 5 sensor categories, and accredited partner-lab panels into one continuous translation of your body.

MEASURED CONTINUOUSLY · 5 CLINICAL-QUALITY SENSORS

The hardware — and the studies behind each signal.

OPTICAL · 100 Hz
PPG — Photoplethysmography
Dual-wavelength (red + infrared) pulse waveform. HR, HRV (RMSSD), SpO2, perfusion, sleep/wake.
THERMAL
NTC Thermistor
Skin-contact wrist temperature. Overnight dip curve + nocturnal nadir.
Wrist-temperature rhythm is an established circadian index4 linked to disease risk5.
MOTION · 25 Hz
6-axis IMU
Accelerometer + gyroscope. Sleep onset, position, posture, movement artefact rejection.
Actigraphy is the validated wearable standard for sleep/wake assessment6.
AUTONOMIC
EDA Electrodes
Two skin electrodes track sweat-gland conductance — direct sympathetic nervous-system readout.
EDA is the established psychophysiological measure of sympathetic arousal7.
BIO-IMPEDANCE
BIS — Bioimpedance Spectroscopy
Multi-frequency impedance for tissue hydration, vascular resistance, barrier stress.
The clinical method behind body-composition & fluid analysis8.

SENSOR HERITAGE · FIRST DESCRIBED → CLINICALLY VALIDATED

Every sensor traces to a documented clinical first.

1888

Electrodermal activity (EDA)

Fere (France) & Tarchanoff (Russia, 1890) establish the two skin-conductance methods still used today. Standardised for research by the Society for Psychophysiological Research7.

1937

Photoplethysmography (PPG)

Alrick Hertzman (Saint Louis University) builds the first photoelectric plethysmograph and coins the term9.

1962

Bioimpedance (BIS)

Thomasset pioneers tissue impedance for total body water; the tetrapolar method is validated by Lukaski (1985) and standardised by ESPEN8.

1965

Circadian body-temperature clock

Jurgen Aschoff & Rutger Wever, Max Planck Institute (Munich), prove the endogenous temperature rhythm in the bunker experiments10. Wrist-temperature index established 20084.

1974

Pulse oximetry (SpO2)

Takuo Aoyagi (Nihon Kohden, Tokyo) derives SpO2 from the PPG pulse — credited with a 40x reduction in anaesthesia deaths2.

1978

Actigraphy (IMU)

Kripke et al. (UC San Diego) validate wrist actigraphy against EEG/PSG with near-perfect agreement for sleep time11; adopted as the clinical standard by the AASM, 20036.

A FOUNDATION OF VALIDATED SCIENCE

From 1888 to today — the research behind every signal.

QUESTIONNAIRE HERITAGE · FIRST PUBLISHED → CLINICAL STANDARD

Six validated instruments. Decades of peer-reviewed evidence.

1983

Perceived Stress Scale (PSS-10) published.

Cohen, Kamarck & Mermelstein develop the most-cited psychological stress measure in clinical literature — used in 50,000+ research studies. Anchors your Brain score.

1988

Gastrointestinal Symptom Rating Scale (GSRS) published.

Svedlund, Sjodin & Dotevall develop the 15-item scale used in nearly every IBS, dyspepsia, and reflux clinical trial since. Anchors your Gut score.

1989

Pittsburgh Sleep Quality Index (PSQI) published.

Buysse, Reynolds, Monk, Berman & Kupfer at the University of Pittsburgh establish a validated sleep-quality questionnaire, widely used in research. Anchors your Recovery score.

1992

Medical Outcomes Study Short-Form 36 (SF-36) published.

Ware & Sherbourne at the RAND Corporation publish the most-cited general health survey in medical literature — used in over 4,000 clinical studies. Anchors your Longevity benchmark.

1997

Bristol Stool Form Scale (BSFS) published.

Heaton & Lewis at the University of Bristol publish the 7-point visual scale that becomes the global standard for assessing gut transit and IBS subtype.

1997

IBS Severity Scoring System (IBS-SSS) published.

Francis, Morris & Whorwell publish the validated 5-question instrument that becomes the primary endpoint in modern IBS clinical trials.

2026

SenseMe integrates all six into one continuous translation system.

SenseMe integrates all six instruments into one continuous translation system — correlating validated questionnaire data with continuous biosignal readings in a single dashboard.

Citations available on request. SenseMe uses each instrument under its original published methodology.

CONFIRMED IN THE LAB · CLIA-CERTIFIED / ISO-15189 · 7 PARTNER LABS

When you want certainty, the labs confirm it.

Optional add-ons, results flow into your scores.

INFLAMMATION · FINGER-PRICK
Blood Panel
8 biomarkers: hsCRP16, vitamin D, zinc, omega-3, ferritin, selenium, homocysteine, TSH. Finger-prick dried-blood-spot method13.
Results in 3-5 days.
SKIN / GUT · DNA SEQUENCING
Skin Microbiome Swab
Species-level 16S rRNA14 diversity + bacteria : fungi ratio. Painless cheek swab. Read against the reference skin-microbiome map17.
myBioma (EU) · Ombre (US) · 7-10 days.
GUT · CLINICAL STOOL TEST
GI Map
PCR15 DNA: pathogens, H. pylori + virulence, commensals, intestinal markers. Bacterial ID by 16S rRNA14.
Cerascreen (EU) · Vibrant Wellness (US) · 10-14 days.
STRESS · IMMUNOASSAY
Cortisol / DHEA-S
Serum or saliva immunoassay12. Confirms what the EDA stress signal infers — the HPA-axis readout.
Accredited partner lab · 3-5 days.
GUT · PCR SEQUENCING
Fungal / Candida
Genus & species-level fungal overgrowth via ITS-region PCR15 18 — invisible to wrist sensors alone.
Accredited partner lab · 10-14 days.

LAB METHOD HERITAGE · FIRST DESCRIBED → CLINICAL STANDARD

Every lab test runs on a method with a documented clinical first.

1960

Radioimmunoassay — hormone measurement

Rosalyn Yalow & Solomon Berson measure picomolar hormone levels in plasma for the first time — the foundation of every cortisol / DHEA-S immunoassay (Yalow, Nobel Prize 1977)12.

1963

Dried blood spot — finger-prick collection

Robert Guthrie & Ada Susi introduce blood-spot sampling for population newborn screening — the method behind today's at-home finger-prick blood panels13.

1977

16S rRNA sequencing — microbiome ID

Carl Woese & George Fox establish 16S ribosomal RNA as the universal bacterial fingerprint — the basis of all skin- and gut-microbiome profiling14.

1985

Polymerase chain reaction (PCR)

Saiki, Mullis et al. publish the first diagnostic application of PCR — the DNA-amplification engine behind GI-Map and fungal panels (Mullis, Nobel Prize 1993)15.

2003

hsCRP inflammation standard

The CDC & American Heart Association set high-sensitivity CRP as the clinical marker for systemic inflammation and cardiovascular risk16.

2009

Human skin-microbiome map

Grice et al. (NIH / NHGRI) publish the first topographical map of the human skin microbiome — the reference for skin-swab diversity scoring17.

2012

ITS fungal barcode & Human Microbiome Project

Schoch et al. establish the ITS region as the universal fungal barcode18; the Human Microbiome Project Consortium defines the healthy human microbiome reference19.

6
Health domains → 1 Holistic score
5
Clinical-quality wrist sensors
122
Band signals
7
Validated clinical instruments
5
Lab panels
88+ biomarkers
30
Cross-domain connections

STANDARDS WE MEET

GDPR-Compliant
By design · Munich-built
CLIA-Certified (US)
Via partner labs
ISO 15189 (EU)
Via partner labs
Physician-Reviewed
Sense clinical team
Clinical-Quality Components
Named, traceable parts
Open Methodology
Every metric traceable to source

What each of those 219 signals & markers actually measures — below.

The science is solid. Now see it in action.

Get your free AI Skin Scan →Reserve your band — $99 deposit →

DATA ARCHITECTURE · GROUND TRUTH MAPPING

Every value. Every source.
Nothing invented.

SenseMe combines 5 sensors, 7 validated instruments, 5 lab panels, AI scan, and skin patch data into 6 domain scores — each signal traceable to its exact biological source.

Wrist Sensors (PPG · EDA · BIS · NTC · IMU)
Validated Questionnaires
Lab Add-On Tests
AI Skin Scan
Skin Patch
Derived / Computed
Skin16 metrics
BIS — Bioimpedance
HOW IT'S MEASUREDBioimpedance measures water and ion movement across the stratum corneum. A weakened barrier loses more water (higher TEWL proxy) — we detect the impedance shift.
WHAT YOU SEEScore 0–100. Personalised baseline range for healthy adults. Drops below baseline flag barrier disruption.
WHY IT MATTERSYour skin barrier is your first immune defense. A weakened barrier predicts breakouts, sensitivity, and inflammation 24–72h before you visibly see them.
HOW IT'S MEASUREDA low-amperage 50–500 kHz electrical current passes through wrist tissue. Higher water content in dermal layers lowers impedance — the reading is baseline-corrected per individual.
WHAT YOU SEEScore 0–100. Your personal hydration baseline is set after 7 days. Drops from baseline flag under-hydration.
WHY IT MATTERSDermal hydration governs skin elasticity, barrier repair speed, and inflammation response. It is one of the most sensitive early signals of systemic dehydration.
HOW IT'S MEASUREDBioimpedance at lower frequencies (50 kHz) is sensitive to tissue fluid shifts associated with sub-dermal inflammation. Compared against your rolling 14-day personal baseline.
WHAT YOU SEERelative index vs. your baseline. Values above baseline indicate elevated sub-dermal fluid and potential inflammatory activity.
WHY IT MATTERSSub-dermal inflammation often precedes visible skin reactions by 12–48 h — catching it early allows intervention before a flare becomes visible.
NTC Thermistor — Skin Temperature
HOW IT'S MEASUREDWrist temperature compared against your rolling 14-day personal baseline.
WHAT YOU SEEDegrees Celsius. Typical wrist range depending on environment. An overnight drop from baseline is expected; smaller drops may indicate stress or illness.
WHY IT MATTERSSkin temperature reflects blood flow, metabolic rate, and nervous system state — changes that precede subjective symptoms by hours.
HOW IT'S MEASUREDA skin-contact thermistor logs wrist temperature overnight. The full curve is analysed for depth, timing, and recovery arc against your personal baseline.
WHAT YOU SEECurve shape score + nadir temperature. A clean overnight dip indicates good thermoregulation. Flat or inverted curves flag poor recovery.
WHY IT MATTERSThe overnight temperature dip drives melatonin rhythm and sleep stage depth. A disrupted curve is one of the earliest signals of impaired recovery.
HOW IT'S MEASUREDDerived from the rate at which your wrist temperature drops at sleep onset and recovers at wake time. Compared against your personal 14-day rolling baseline.
WHAT YOU SEEEfficiency score 0–100. Higher scores indicate faster, deeper thermoregulatory response. Lower scores correlate with poor sleep quality.
WHY IT MATTERSThermoregulation efficiency is tightly coupled to autonomic nervous system function — a falling score over weeks can indicate chronic stress or hormonal drift.
PPG · IMU
HOW IT'S MEASUREDInfrared and red PPG light through the skin measures pulsatile blood volume at the wrist. The amplitude and shape of the pulse waveform reflect peripheral vascular tone and microvascular perfusion.
WHAT YOU SEEIndex 0–100. Your baseline is set after 14 days. Lower values suggest reduced peripheral perfusion; values that drop suddenly may indicate vasoconstriction from stress or cold.
WHY IT MATTERSPeripheral microcirculation is a direct readout of how well blood reaches your skin. Poor perfusion slows barrier repair, reduces nutrient delivery, and accelerates visible ageing.
HOW IT'S MEASUREDPPG-derived sleep staging (light, deep, REM) is combined with IMU movement data to compute the total time spent in slow-wave sleep — the phase when skin cell turnover and barrier repair peak.
WHAT YOU SEEMinutes in deep/repair sleep per night. Consistently low deep-sleep time signals insufficient skin repair time.
WHY IT MATTERSSkin regeneration is almost entirely nocturnal and deep-sleep-gated. This metric connects your sleep architecture directly to your Skin score.
HOW IT'S MEASUREDThe IMU's 6-axis accelerometer + gyroscope (sampling at 25 Hz) tracks your dominant sleep position and how much time you spend in face-down or side-compression orientations.
WHAT YOU SEEDominant position + compression time (minutes/night). Prolonged consistent single-side compression is flagged as elevated mechanical stress.
WHY IT MATTERSChronic sleep-position pressure accelerates collagen breakdown and contributes to asymmetric skin ageing. Most trackers ignore this signal entirely.
Questionnaires
HOW IT'S MEASUREDScored using the SenseMe Skin Baseline questionnaire — a short self-report instrument designed by the Sense clinical team, calibrated against published reference populations. Completed once at onboarding, updatable quarterly.
WHAT YOU SEEFitzpatrick type I–VI + Baumann skin type (Oily/Dry/Combination/Sensitive overlay). Used as a fixed calibration factor across all Skin domain scores.
WHY IT MATTERSSkin type fundamentally shifts what 'normal' looks like for every other Skin metric. A score calibrated for Type II skin is meaningless applied to Type V.
HOW IT'S MEASUREDScored using the SenseMe Skin Baseline questionnaire — a daily self-report log of morning and evening skincare steps, calibrated against published research on routine adherence and barrier outcomes.
WHAT YOU SEEScore 0–100 (weekly average). Consistent high adherence is healthy. Sustained drops are flagged as a confounding variable for Skin score interpretation.
WHY IT MATTERSRoutine adherence is one of the strongest predictors of skin barrier health. Knowing it lets SenseMe separate what your skin is doing from what you're doing to it.
Lab Add-Ons
HOW IT'S MEASUREDSkin swab sample shipped to a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. DNA sequencing identifies microbial species abundance and diversity on the skin surface.
WHAT YOU SEEShannon diversity index. Lower values suggest dysbiosis. Recommended twice yearly.
WHY IT MATTERSThe skin microbiome is a frontline regulator of barrier integrity and immune response. Low diversity is associated with eczema, acne, and accelerated skin ageing.
HOW IT'S MEASUREDPhysical sample processed at a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. The skin patch also provides continuous local pH readings in direct skin contact.
WHAT YOU SEEpH value. Elevated surface pH indicates compromised acid mantle and elevated bacterial proliferation risk.
WHY IT MATTERSSkin surface pH is a master regulator of barrier enzyme activity and microbiome balance. Small shifts meaningfully alter barrier function.
AI Skin Scan
WHAT YOU SEEComposite score 0–100. Updates each time you scan (recommended weekly).
Skin Patch
HOW IT'S MEASUREDAdhesive multi-sensor patch worn for 24h that measures local trans-epidermal water loss (TEWL), pH, and hydration in direct skin contact. Patch data syncs to your SenseMe ID after removal.
WHAT YOU SEETEWL in g/m²h, pH, and hydration in Corneometer units, each shown against healthy reference ranges. 24h continuous trace.
WHY IT MATTERSThe patch provides the most direct measurement of barrier function available outside a clinical dermatology lab — no proxy, no inference.
HOW IT'S MEASUREDComputed from the delta between patch-measured TEWL and BIS-derived barrier stress over the preceding 48h. A widening gap between the two signals indicates accelerating barrier breakdown.
WHAT YOU SEERisk score 0–100. Elevated scores trigger a 'Flare Alert' in the app with recommended immediate interventions.
WHY IT MATTERSBy combining two independent barrier signals, Flare Risk gives 24–72h advance warning of skin reactions — turning a reactive skincare routine into a proactive one.
Gut12 metrics
EDA / GSR — Gut-Brain Axis
HOW IT'S MEASUREDTwo skin-contact electrodes track sweat-gland-driven conductance changes via electrodermal activity (EDA). The gut-brain axis generates characteristic EDA patterns distinct from cortical stress — identified by timing, duration, and recovery shape.
WHAT YOU SEEAmplitude in microsiemens (μS). Individual baselines vary widely. SenseMe uses personalised deviation rather than population thresholds.
WHY IT MATTERSThe enteric nervous system is often called the 'second brain'. EDA picks up visceral stress that you may not consciously register — often preceding digestive symptoms by hours.
HOW IT'S MEASUREDEDA phasic responses are time-correlated with HRV (PPG) and movement data (IMU) to compute the coherence between your gut nervous system signals and your central nervous system state.
WHAT YOU SEEIndex 0–100. Higher coherence indicates a well-regulated gut-brain axis. Chronically low scores correlate with dysbiosis and IBS-type symptom patterns.
WHY IT MATTERSThe gut-brain axis governs mood, immunity, and metabolic regulation. Coherence is a systems-level signal that no single sensor can provide alone.
PPG · IMU — Digestive Signals
HOW IT'S MEASUREDPPG-derived HRV is measured continuously. SenseMe identifies the 90-minute post-meal window (from self-logged meal time or IMU-detected eating posture) and quantifies the typical parasympathetic shift.
WHAT YOU SEERMSSD drop in ms vs. pre-meal baseline. A moderate drop is normal digestion. Larger drops suggest high digestive load or inflammatory response.
WHY IT MATTERSYour heart rate variability reveals how much metabolic resources digestion is consuming. Consistently large suppression signals poor food tolerance or chronic gut stress.
HOW IT'S MEASUREDThe IMU's 6-axis accelerometer detects micro-movements and rhythmic patterns at 25 Hz. Abdominal breathing and peristaltic movement create distinctive low-frequency signal signatures.
WHAT YOU SEEPattern quality score 0–100. Your personal baseline is set after 7 days of wear. Deviations from baseline flag altered motility.
WHY IT MATTERSAltered abdominal movement patterns are detectable days before subjective symptoms — providing early warning of dysbiosis, inflammation, or dietary intolerance flares.
HOW IT'S MEASUREDIMU data overnight identifies the characteristic low-amplitude cyclic movements of the migrating motor complex (MMC) — the gut's nocturnal housekeeping contractions.
WHAT YOU SEEMMC cycle detection: present / partial / absent. Healthy individuals show multiple complete cycles per night. Sustained absence over several nights flags impaired gut motility.
WHY IT MATTERSThe migrating motor complex is critical for gut microbiome balance and overnight bacterial clearance. Loss of MMC activity is an early sign of imbalance in overnight gut bacterial patterns.
HOW IT'S MEASUREDDerived from IMU colonic movement patterns combined with Bristol Stool Form Scale daily entries. The algorithm correlates motility signals with stool form to estimate colonic transit time.
WHAT YOU SEEEstimated transit time in hours. Shorter times indicate fast transit; longer times indicate slow transit / constipation.
WHY IT MATTERSTransit time is a master variable for gut health — too fast means inadequate nutrient absorption; too slow means bacterial overgrowth risk and elevated toxin reabsorption.
Questionnaires — Daily + Monthly
HOW IT'S MEASUREDOne-tap visual selection on the 7-point Bristol scale validated by Heaton & Lewis (1997). Logged daily, trended over 7-day windows.
WHAT YOU SEEType 1–7. Types 3–5 typical for healthy transit. Types 1–2 indicate slow transit; 6–7 indicate fast transit.
WHY IT MATTERSStool form is the most direct, daily-measurable signal of gut transit time, hydration, and fibre adequacy — the foundation of gut-skin axis interpretation.
HOW IT'S MEASUREDScored using the validated GSRS instrument. 15 items across 5 subscales (Reflux, Abdominal Pain, Indigestion, Diarrhoea, Constipation) using the original published methodology.
WHAT YOU SEETotal score 15–105. Higher scores indicate more severe symptoms. Elevated scores flag significant GI burden. Completed monthly.
WHY IT MATTERSThe GSRS separates which GI symptom cluster you experience — enabling SenseMe to route you to the right intervention rather than generic gut advice.
HOW IT'S MEASUREDScored using the validated IBS-SSS questionnaire. 5 items including pain severity, frequency, bloating, satisfaction, and interference with life. Published methodology by Francis et al.
WHAT YOU SEEScore 0–500. Higher scores indicate more severe IBS. Completed monthly, trended over quarters.
WHY IT MATTERSIBS-SSS is the most widely validated clinical instrument for IBS severity tracking — connecting your subjective gut experience to objective sensor and lab data.
HOW IT'S MEASUREDShort self-report instrument designed by the Sense clinical team, calibrated against published reference populations. Covers dietary patterns, food sensitivities, stress-gut relationship, and previous diagnoses.
WHAT YOU SEEQualitative profile used as calibration context. Updated quarterly or when lifestyle changes significantly.
WHY IT MATTERSThe Gut Baseline anchors all Gut domain scores to your individual context — a person with IBS-C and a person with IBS-D have radically different 'normal' baselines.
GI MAP
HOW IT'S MEASUREDStool sample shipped to partner lab (Cerascreen EU / Vibrant Wellness US). DNA sequencing identifies pathogens, opportunistic microbes, and commensal bacteria abundance.
WHAT YOU SEEPer-species abundance values + clinical reference ranges + 7 H. pylori virulence factors.
WHY IT MATTERSThe gold-standard view of your gut microbiome. Recommended quarterly — connects directly to your Skin, Brain, and Recovery scores.
HOW IT'S MEASUREDStool sample processed at a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. PCR-based DNA sequencing identifies Candida species and other fungal organisms at the genus and species level.
WHAT YOU SEEPer-species abundance vs. clinical reference ranges. Elevated Candida albicans is flagged as potential overgrowth.
WHY IT MATTERSFungal overgrowth is a common but underdiagnosed driver of chronic bloating, fatigue, and skin flares — invisible to wrist sensors alone.
Brain12 metrics
EDA / GSR — Stress Engine
HOW IT'S MEASUREDTwo skin-contact electrodes track sweat-gland-driven conductance changes. Picks up sympathetic nervous system activation within seconds. Phasic spikes are separated from tonic (background) level.
WHAT YOU SEEConductance in μS. Phasic events above baseline counted as SNS activations. Daily count + peak amplitude reported.
WHY IT MATTERSDirect measurement of your fight-or-flight nervous system — not inferred from heart rate. The most real-time stress signal available outside a clinical lab.
HOW IT'S MEASUREDEDA phasic spike detection algorithm counts distinct sympathetic activation events over a 24h window. Brief events followed by recovery are classified as discrete stress events.
WHAT YOU SEEEvent count per day. Higher counts consistently correlate with elevated cortisol and impaired recovery.
WHY IT MATTERSFrequency matters as much as intensity. High event counts with low amplitude often indicate a chronically activated nervous system — harder to detect with intensity metrics alone.
HOW IT'S MEASUREDThe maximum EDA phasic response amplitude recorded within a 24h window, measured in microsiemens above tonic baseline. Sampled continuously and corrected for motion artefact via IMU.
WHAT YOU SEEPeak amplitude in μS above baseline. Personalised threshold after 14 days. Readings well above your baseline flag extreme activation events.
WHY IT MATTERSPeak amplitude identifies acute high-intensity stress moments — often correlating with specific life events that the daily average would smooth over.
HOW IT'S MEASUREDAfter each detected EDA spike, the rate of conductance return to tonic baseline is modelled as an exponential decay curve. The time constant (τ) captures how quickly your nervous system recovers.
WHAT YOU SEERecovery time constant in seconds. Faster recovery indicates resilient autonomic response. Consistently slow recovery flags autonomic dysregulation.
WHY IT MATTERSRecovery speed predicts cumulative stress load better than peak amplitude. A nervous system that takes several minutes to recover from minor stress is chronically over-stressed.
HOW IT'S MEASUREDThe slow-changing background (tonic) EDA level is tracked over 24h and 7-day windows. Drift in the tonic level — independent of phasic events — reflects longer-term sympathetic tone changes.
WHAT YOU SEETonic drift in μS/day vs. your 14-day rolling average. Sustained upward drift over 7 days flags chronic stress accumulation.
WHY IT MATTERSTonic drift is invisible in single-point measurements. Only continuous wrist monitoring reveals the slow creep of chronic stress that precedes burnout.
PPG · NTC · IMU
HOW IT'S MEASUREDInfrared and red PPG light measures beat-to-beat heart intervals at 100 Hz. HRV (RMSSD) is computed and cross-correlated with same-minute EDA readings to assess parasympathetic withdrawal during stress.
WHAT YOU SEECorrelation coefficient (−1 to +1). A strong negative correlation between EDA spikes and HRV dips confirms a healthy stress-response coupling.
WHY IT MATTERSHRV alone can't distinguish stress from exercise. Cross-correlating with EDA isolates the autonomic stress response — making the Brain score resistant to physical activity confounds.
HOW IT'S MEASUREDPPG-derived heart rate during confirmed resting periods (IMU-verified stillness, overnight). 7-day rolling average plotted against your personal baseline established in the first 14 days.
WHAT YOU SEEBeats per minute (bpm). Your personal resting HR baseline. Increases above your 7-day average flag elevated stress or early illness.
WHY IT MATTERSResting heart rate is one of the most reliable long-term markers of autonomic fitness and chronic stress load — and one of the earliest signs of overtraining or immune activation.
HOW IT'S MEASUREDNTC thermistor data is time-correlated with EDA phasic events. During sympathetic activation, peripheral vasoconstriction causes a measurable skin temperature drop at the wrist within 30–60 seconds.
WHAT YOU SEETemperature drop in °C per stress event. A modest drop is a typical stress response. Absence of temperature response may indicate desensitised autonomic response.
WHY IT MATTERSSkin temperature during stress confirms that the EDA signal reflects true sympathetic output — not sensor artefact. It adds a second independent channel to stress detection.
HOW IT'S MEASUREDThe IMU's 6-axis accelerometer identifies movement episodes. EDA and HRV readings during active movement are flagged and excluded from Brain score computation to prevent exercise-induced false positives.
WHAT YOU SEEFilter active/inactive per minute. A portion of daily EDA readings are excluded due to movement in typical users.
WHY IT MATTERSWithout the IMU confound filter, stress scores would spike every time you climbed stairs. This is what separates a real stress metric from a glorified step counter.
Questionnaires + Screen Time API
HOW IT'S MEASUREDValidated 10-item questionnaire developed by Cohen, Kamarck & Mermelstein (1983). The most widely used psychological stress measure in research. Self-reported monthly.
WHAT YOU SEETotal score 0–40. Higher scores indicate greater perceived stress. Trended month-over-month.
WHY IT MATTERSSubjective perception is irreplaceable. PSS-10 anchors what the EDA sensor measures against how you actually feel — calibrating your Brain score against decades of clinical research.
HOW IT'S MEASUREDiOS Screen Time or Android Digital Wellbeing API data (with your permission) provides daily app-usage duration by category. SenseMe analyses late-night usage, social-media time, and total daily screen exposure.
WHAT YOU SEEDaily screen time in hours by category. Baseline established over 14 days. Late-night usage is flagged as sleep-onset interference.
WHY IT MATTERSBehavioral data closes the loop between stress signals and lifestyle causes. Knowing your EDA spiked at 23:00 means more with screen-time context than without it.
Hormone Panel
HOW IT'S MEASUREDBlood or saliva sample shipped to a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. Serum cortisol and DHEA-S (dehydroepiandrosterone-sulfate) are measured by immunoassay.
WHAT YOU SEECortisol in nmol/L, DHEA-S in μmol/L. Cortisol:DHEA-S ratio is the key output. An elevated ratio indicates HPA axis imbalance. Recommended twice yearly.
WHY IT MATTERSThe cortisol:DHEA-S ratio is the most clinically validated hormonal marker of chronic stress and adrenal function — anchoring your Brain score in biochemistry.
Recovery12 metrics
PPG
HOW IT'S MEASUREDBeat-to-beat heart interval variation, calculated overnight when your body is at rest. Root Mean Square of Successive Differences (RMSSD) — a widely used measure of vagal tone.
WHAT YOU SEEMilliseconds. Personal baseline is set after 14 nights. Daily reading shown as deviation from your own norm — not population averages.
WHY IT MATTERSHRV is the single most predictive metric for next-day readiness, immune resilience, and long-term healthspan. Lower than baseline = body in repair mode.
HOW IT'S MEASUREDPPG heart rate variability patterns combined with IMU movement data are fed into a validated sleep-staging algorithm. Light, deep (slow-wave), and REM stages are identified.
WHAT YOU SEEMinutes per stage per night. Deep, REM, and light stages are each reported.
WHY IT MATTERSTotal sleep time tells you how long you slept. Sleep architecture tells you whether that sleep was actually restorative — a completely different question.
HOW IT'S MEASUREDPPG red and infrared light absorption ratios measure blood oxygen saturation (SpO₂) continuously overnight. The lowest reading during the sleep period is recorded as the nocturnal nadir.
WHAT YOU SEEPercentage (%). A healthy nadir stays high. Lower overnight readings suggest reduced oxygen levels and, if they persist, are worth discussing with a healthcare professional.
WHY IT MATTERSNocturnal SpO₂ dips are an important overnight signal — invisible without continuous monitoring — and worth raising with a healthcare professional if they persist.
IMU · NTC · BIS
HOW IT'S MEASUREDIMU accelerometer detects the transition from waking movement to stillness-onset. SenseMe defines sleep onset as a sustained period of near-zero movement following an 'in bed' posture detection.
WHAT YOU SEEMinutes from 'in bed' to sleep onset. Consistently long latency suggests you are taking longer than usual to fall asleep.
WHY IT MATTERSSleep latency is a direct readout of evening cortisol and racing-mind arousal. Tracking it nightly reveals which daytime behaviours accelerate or delay your wind-down.
HOW IT'S MEASUREDIMU-detected sleep onset to final wake event, summing all sleep periods including brief naps detected during the day. Cross-validated against PPG heart rate patterns.
WHAT YOU SEEHours and minutes. SenseMe compares against a healthy adult range. Consistently short or long sleep is flagged as a recovery risk.
WHY IT MATTERSTotal sleep time is the foundation of the Recovery domain — every other recovery metric is interpreted in the context of whether you slept enough total hours.
HOW IT'S MEASUREDNTC thermistor logs wrist temperature overnight. The minimum temperature reached during the sleep period is recorded and compared against your 14-day rolling personal baseline.
WHAT YOU SEETemperature nadir in °C. A clear drop from pre-sleep baseline is normal. Smaller drops indicate suboptimal thermoregulation and typically correlate with lighter sleep.
WHY IT MATTERSCore temperature must drop for deep sleep to initiate. The nadir depth predicts slow-wave sleep duration better than any questionnaire.
HOW IT'S MEASUREDA low-amperage 50–500 kHz electrical current passes through wrist tissue. The impedance reading at multiple frequencies is decomposed to estimate intracellular vs. extracellular fluid balance — a proxy for overall hydration and lean mass status.
WHAT YOU SEEHydration index relative to your personal baseline. Values well below baseline flag dehydration; values well above may indicate fluid retention or inflammation.
WHY IT MATTERSHydration status directly modulates muscle recovery, cognitive function, and cardiovascular load. Catching dehydration before it becomes symptomatic is one of the simplest ways to improve daily performance.
Questionnaires Daily + Monthly
HOW IT'S MEASUREDScored using the SenseMe daily self-report instrument — a 3-item morning prompt (energy, mood, body feel) designed to capture subjective readiness in seconds. Calibrated against published reference populations.
WHAT YOU SEEScore 0–10. Your personal baseline established after 14 days. Low readings consistently trigger a lower training-intensity recommendation.
WHY IT MATTERSSubjective readiness consistently predicts performance and injury risk better than any single sensor metric. SenseMe uses it to anchor the objective data in how you actually feel.
HOW IT'S MEASUREDScored using the SenseMe daily Sleep Log — a single morning retrospective rating of sleep quality. Completed before checking objective sensor data to avoid anchoring bias.
WHAT YOU SEEScore 1–10. 7-day rolling average. A divergence between subjective score and PPG-derived sleep score flags a perception-reality gap worth investigating.
WHY IT MATTERSThe gap between how you slept and how you feel you slept is itself meaningful — it often reflects stress, low mood, or a mismatch between how restful sleep felt and what was measured.
HOW IT'S MEASUREDValidated 19-item self-report instrument developed by Buysse et al., University of Pittsburgh (1989). A validated sleep-quality questionnaire, widely used in research. Filled monthly.
WHAT YOU SEEGlobal score 0–21. Lower scores indicate good sleep quality. Trended over months, anchored against your wristband's objective sleep staging.
WHY IT MATTERSSensor data tells you how you slept. PSQI tells you how you felt about how you slept. The combination is what matters — your perception drives your behavior.
Blood Panel
HOW IT'S MEASUREDFinger-prick blood spot shipped to a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. Serum ferritin and 25-hydroxyvitamin D are measured by immunoassay.
WHAT YOU SEEFerritin in ng/mL and Vitamin D in nmol/L, each shown against its optimal range. Both are applied as multipliers to your Recovery domain score.
WHY IT MATTERSLow ferritin and Vitamin D are the two most common, most fixable, and most frequently missed drivers of chronic fatigue and poor sleep quality.
Heart9 metrics
PPG — Cardiovascular
HOW IT'S MEASUREDInfrared and red PPG light measures pulsatile blood volume at the wrist continuously. Resting heart rate is extracted from confirmed rest periods (IMU-verified stillness) and averaged over a 7-day rolling window.
WHAT YOU SEEBeats per minute (bpm). Baselines differ widely between trained and sedentary adults. SenseMe uses your personal baseline rather than population norms.
WHY IT MATTERSResting heart rate is one of the most robust long-term cardiovascular health indicators. A declining resting HR over months reliably reflects improving cardiovascular fitness.
HOW IT'S MEASUREDSame calculation as Recovery's HRV but evaluated against cardiovascular reference ranges by age and sex.
WHAT YOU SEEMilliseconds. Personalised reference range. Trend over 90 days for true cardiac fitness signal.
WHY IT MATTERSLow HRV is one of the strongest predictors of cardiovascular events and all-cause mortality in epidemiological studies — even beyond resting heart rate.
HOW IT'S MEASUREDStandard deviation of all NN (normal-to-normal beat) intervals overnight. Captures total autonomic variability including both sympathetic and parasympathetic contributions, sampled at 100 Hz via PPG.
WHAT YOU SEEMilliseconds. Healthy ranges shift with age. SenseMe uses age- and sex-adjusted personalised baselines.
WHY IT MATTERSSDNN captures long-range autonomic variability that RMSSD misses — it is the measure used in most large-scale cardiovascular mortality prediction studies.
HOW IT'S MEASUREDSenseMe detects postural transitions from lying to standing via IMU. The PPG-measured heart rate response in the first 30 seconds is compared against your 14-day personal baseline.
WHAT YOU SEEHR rise in bpm within 30s of standing. Both an excessive rise and a blunted rise flag autonomic dysregulation.
WHY IT MATTERSOrthostatic heart rate response is a sensitive daily marker of autonomic nervous system health, dehydration, and early cardiovascular dysfunction — captured passively every morning.
HOW IT'S MEASUREDPulse transit time is derived from the time delay between each heartbeat (PPG R-wave) and the corresponding wrist pulse arrival, combined with BIS-derived vascular resistance. Correlates with arterial blood pressure changes.
WHAT YOU SEETransit time in milliseconds. Shorter PTT correlates with higher arterial blood pressure. Personalised baseline; trend matters more than absolute value.
WHY IT MATTERSPTT provides a continuous, cuffless blood pressure trend — not a clinical blood pressure reading, but a directional signal that flags rising vascular resistance before it becomes symptomatic.
BIS · NTC — Cellular + Thermal
HOW IT'S MEASUREDA low-amperage 50–500 kHz electrical current passes through wrist tissue. The impedance profile at frequencies sensitive to vascular tissue reveals changes in vessel wall stiffness and resistance.
WHAT YOU SEEIndex relative to your personal baseline. Elevated resistance over multiple days flags increasing arterial stiffness.
WHY IT MATTERSArterial stiffness is an independent predictor of cardiovascular risk. BIS-based vascular resistance tracking provides a daily signal that connects your Heart score to structural vascular health.
HOW IT'S MEASUREDNTC wrist temperature and PPG heart rate are cross-correlated over 24h windows. Healthy thermoregulation produces a predictable inverse relationship between core temperature and heart rate during overnight recovery.
WHAT YOU SEECoupling coefficient (−1 to +1). Healthy coupling is strongly negative. Decoupling (coefficient approaching 0) over multiple nights flags autonomic dysregulation or inflammation.
WHY IT MATTERSTemperature-heart rate coupling is a systems-level signal invisible to either sensor alone. Decoupling often precedes illness, overtraining, or metabolic stress by 24–48h.
Cardio Baseline Questionnaire
HOW IT'S MEASUREDScored using the SenseMe Cardio Baseline questionnaire — a short self-report instrument designed by the Sense clinical team covering exercise history, breathlessness patterns, and cardiovascular symptoms. Completed at onboarding and updated quarterly.
WHAT YOU SEEQualitative profile + estimated VO₂ max tier (Low / Moderate / High / Athletic). Used as calibration context for Heart domain score interpretation.
WHY IT MATTERSCardio fitness level fundamentally changes what 'normal' looks like for HRV and resting heart rate. A 72 bpm resting HR means something very different for a sedentary person vs. a trained cyclist.
Blood Panel
HOW IT'S MEASUREDFinger-prick blood spot or venous draw, shipped to partner lab (Medichecks EU / EverlyWell US). Measures total cholesterol, LDL, HDL, triglycerides, hs-CRP inflammation marker.
WHAT YOU SEEEach value with personalised reference range. Flags optimal vs low-normal vs out-of-range.
WHY IT MATTERSThe cardiovascular markers that decades of clinical research show actually move outcomes. SenseMe connects them directly to your live HRV trend — full feedback loop.
Longevity10 metrics
Cross-Domain Computation
HOW IT'S MEASUREDComputed from the current Brain domain score (stress, sleep cognition, HRV stress correlation). Applied as the largest single contributor in the longevity algorithm, reflecting the primacy of chronic stress in biological ageing research.
WHAT YOU SEEWeighted contribution 0–100. Updated daily as Brain domain score updates.
WHY IT MATTERSChronic psychological stress is the strongest modifiable driver of biological age acceleration. Epigenetic clock research consistently identifies stress-related methylation patterns as top ageing accelerants.
HOW IT'S MEASUREDComputed from the current Gut domain score (microbiome proxies, motility, inflammation). Applied as a weighted contributor in the longevity algorithm, reflecting the gut's role as a systemic inflammation and metabolite regulator.
WHAT YOU SEEWeighted contribution 0–100. Updated daily.
WHY IT MATTERSGut microbiome diversity is one of the most reliable differentiators between healthy and unhealthy ageing cohorts. A dysbiotic gut accelerates biological age through systemic low-grade inflammation.
HOW IT'S MEASUREDComputed from the current Skin domain score (barrier integrity, hydration, microcirculation). Applied as a weighted contributor, reflecting skin as both a direct ageing proxy and a systemic health mirror.
WHAT YOU SEEWeighted contribution 0–100. Updated daily.
WHY IT MATTERSSkin biological age correlates strongly with chronological age across populations. More importantly, it responds rapidly to lifestyle interventions — making it a sensitive short-term feedback signal.
HOW IT'S MEASUREDComputed from the Heart domain's HRV RMSSD and SDNN trends. Functions as a positive modifier to the longevity algorithm — high HRV relative to age-sex norms reduces the computed biological age.
WHAT YOU SEEBonus modifier reducing biological age. Triggered when HRV RMSSD stays high relative to your age/sex group for multiple consecutive days.
WHY IT MATTERSHRV is the only wearable-accessible metric with robust longitudinal evidence for all-cause mortality prediction. The bonus acknowledges that cardiovascular resilience deserves extra weight in longevity scoring.
Computed Outputs
HOW IT'S MEASUREDComputed from weighted contributions of the Brain, Gut, and Skin scores, with an HRV bonus modifier. Lab add-ons increase confidence (a GI MAP unlocks full gut weighting).
WHAT YOU SEEYears (e.g. "biological age 42.3, 4 years younger than chronological"). Updates daily.
WHY IT MATTERSBiological age moves on weeks-to-months timescales — fast enough to give you actionable feedback on lifestyle changes, slow enough to be meaningful.
HOW IT'S MEASUREDComputed from the 12-week trajectory of your biological age, compared against the expected pace where biological and calendar time move in step.
WHAT YOU SEEA multiplier showing whether you are aging slower or faster than calendar time, tracked as a 12-week trend.
WHY IT MATTERSBio age tells you where you are. Pace tells you which direction you're moving — the only metric that captures whether your current life is adding or removing years.
HOW IT'S MEASUREDComputed from the 12-week linear regression of biological age estimate values. A negative slope means biological age is declining (ageing slower than chronological time).
WHAT YOU SEETrend direction (improving / stable / declining) + rate in months of biological age change per calendar month.
WHY IT MATTERSA single biological age reading is a snapshot. The trajectory tells you whether your interventions are working — typically requiring several weeks to show meaningful movement.
Profile Input
HOW IT'S MEASUREDEntered at profile setup and updated automatically on your birthday. Used as the reference baseline against which your biological age estimate is compared.
WHAT YOU SEEYears. No range — this is your actual calendar age. All longevity metrics are expressed as a delta relative to this value.
WHY IT MATTERSBiological age is meaningless without the chronological reference. Knowing you're biologically 38 only matters in the context of knowing your actual age is 44.
Lab Add-Ons
HOW IT'S MEASUREDPhysical sample processed at a CLIA-certified (US) or ISO-15189 accredited (EU) partner lab. Combines skin microbiome Shannon diversity index (swab) with serum cortisol (blood spot) to produce a composite longevity modifier.
WHAT YOU SEEComposite modifier applied to Longevity score. A healthy combined profile increases confidence in the biological age estimate.
WHY IT MATTERSThe combination of microbiome diversity and cortisol captures both the environmental and hormonal ageing axes in a single lab add-on — the highest-ROI lab test in the Longevity domain.
HOW IT'S MEASUREDGI MAP stool sequencing result is used to upgrade the Gut domain's confidence weighting in the Longevity algorithm from a proxy-based estimate to a direct-measurement value.
WHAT YOU SEEConfidence upgrade: without a GI MAP, gut weighting relies on sensor/questionnaire proxies; with a GI MAP, full gut weighting is unlocked.
WHY IT MATTERSWithout stool sequencing, the gut component of biological age is an estimate. GI MAP converts it to a measured value — materially changing the precision of your biological age calculation.

The stack

Every signal accounted for.

SenseMe doesn't rely on one data source. It triangulates across many — most validated by decades of clinical research. Here is the science under the hood.

Continuous
Sensors
5 sensors
EDA · PPG · 6-axis IMU · BIS · NTC.
Daily
60 seconds every morning
Daily recovery self-report
How you slept, how you feel, what you noticed. A short tap-based check-in calibrates the sensor data against your own perception — the only signal a wristband can't measure.
Monthly · Clinically validated
SenseMe in-house clinical questionnaire covering barrier function, sensitivity, hormonal cycle phase, and product use. Updates your Skin sub-scores monthly.
Developed by the Sense clinical team
The most-cited general health survey in medical literature — used in over 4,000 clinical studies. Anchors your Longevity and Recovery scores against decades of validated benchmarks.
Developed by John E. Ware Jr. & Cathy Donald Sherbourne, RAND Corporation, 1992
A validated stool-form scale, widely used in research, for assessing gut transit time and IBS subtype classification — a 7-point visual scale that turns a subjective observation into a comparable, trackable signal. Used in nearly every gut clinical trial since the late 1990s.
Developed by Dr. Ken W. Heaton & Dr. Stephen J. Lewis, University of Bristol, 1997
Validated 5-question instrument scoring abdominal pain severity, frequency, bloating, bowel dissatisfaction and quality-of-life impact. The primary endpoint in modern IBS clinical trials — and now a continuous input to your Gut score.
Developed by C.Y. Francis, J. Morris & P.J. Whorwell, 1997
15-item validated scale tracking reflux, abdominal pain, indigestion, diarrhea and constipation syndromes. Used in clinical trials for IBS, dyspepsia, and reflux disease.
Developed by Jan Svedlund, Inger Sjödin & Göran Dotevall, 1988
The most widely used psychological stress measure in the world — 10 items measuring how unpredictable, uncontrollable and overloaded you've felt in the past month. Anchors your Brain score.
Developed by Sheldon Cohen, Tom Kamarck & Robin Mermelstein, 1983
19 items measuring subjective sleep quality, latency, duration, efficiency, disturbances, medication use and daytime dysfunction. Validates the wristband's sleep staging against your lived experience.
Developed by Daniel J. Buysse, Charles F. Reynolds III, Timothy H. Monk, Susan R. Berman & David J. Kupfer, University of Pittsburgh, 1989
On-demand
When you take it
AI Skin Scan
Real-time facial analysis: barrier integrity, hydration, sensitivity, pigmentation. Matches against brands of skincare and supplements.
See the AI Skin Scan spotlight above ↑
Quarterly · Optional
Optional · Quarterly
Lab add-on tests
Skin Microbiome · GI Map · Blood Panel · Hormone Panel · pH Strips. Each result syncs directly into your pillar scores via your SenseMe ID — no second app, no PDFs.
See lab add-ons below ↓

0 signals across 5 tiers. 0 validated clinical instruments. 1 dashboard that finally listens to all of them.

This is why your data finally makes sense.

REFERENCES

Sensor References — Peer-Reviewed

  1. 1.Schafer A, Vagedes J. (2013) How accurate is pulse rate variability as an estimate of heart rate variability? Int J Cardiol 166(1):15-29. https://www.sciencedirect.com/science/article/abs/pii/S0167527312003269
  2. 2.Nitzan M, Romem A, Koppel R. (2014) Pulse oximetry: fundamentals and technology update. Med Devices (Auckl) 7:231-9. https://pubmed.ncbi.nlm.nih.gov/25031547/
  3. 3.Chinoy ED, et al. (2021) Performance of seven consumer sleep-tracking devices compared with polysomnography. Sleep 44(5):zsaa291. https://academic.oup.com/sleep/article/44/5/zsaa291/6055610
  4. 4.Sarabia JA, et al. (2008) Circadian rhythm of wrist temperature in normal-living subjects. Physiol Behav 95(4):570-80. https://pubmed.ncbi.nlm.nih.gov/18761026/
  5. 5.Weston SJ, et al. (2023) Diurnal rhythms of wrist temperature are associated with future disease risk in the UK Biobank. Nat Commun 14:5172. https://www.nature.com/articles/s41467-023-40977-5
  6. 6.Ancoli-Israel S, et al. (2003) The role of actigraphy in the study of sleep and circadian rhythms. Sleep 26(3):342-392. https://academic.oup.com/sleep/article-abstract/26/3/342/2708388
  7. 7.Boucsein W, et al. (2012) Publication recommendations for electrodermal measurements. Psychophysiology 49:1017-1034. https://pubmed.ncbi.nlm.nih.gov/22680988/
  8. 8.Kyle UG, et al. (2004) Bioelectrical impedance analysis — Parts I & II (ESPEN guidelines). Clin Nutr 23:1226-1243 & 1430-1453. https://pubmed.ncbi.nlm.nih.gov/15556267/
  9. 9.Hertzman AB. (1937) Photoelectric plethysmography of the fingers and toes in man. Proc Soc Exp Biol Med 37:529-534. https://journals.sagepub.com/doi/abs/10.3181/00379727-37-9630
  10. 10.Aschoff J. (1965) Circadian rhythms in man. Science 148(3676):1427-1432. https://www.science.org/doi/10.1126/science.148.3676.1427
  11. 11.Kripke DF, et al. (1978) Wrist actigraphic measures of sleep and rhythms. Electroencephalogr Clin Neurophysiol 44(5):674-676. https://www.sciencedirect.com/science/article/abs/pii/0013469478901335

Lab Method References — Peer-Reviewed

  1. 12.Yalow RS, Berson SA. (1960) Immunoassay of endogenous plasma insulin in man. J Clin Invest 39(7):1157-1175. https://pubmed.ncbi.nlm.nih.gov/13846364/
  2. 13.Guthrie R, Susi A. (1963) A simple phenylalanine method for detecting phenylketonuria in large populations of newborn infants. Pediatrics 32:338-343. https://pubmed.ncbi.nlm.nih.gov/14063511/
  3. 14.Woese CR, Fox GE. (1977) Phylogenetic structure of the prokaryotic domain: the primary kingdoms. PNAS 74(11):5088-5090. https://pubmed.ncbi.nlm.nih.gov/270744/
  4. 15.Saiki RK, Mullis KB, et al. (1985) Enzymatic amplification of beta-globin genomic sequences and restriction site analysis for diagnosis of sickle cell anemia. Science 230(4732):1350-1354. https://pubmed.ncbi.nlm.nih.gov/2999980/
  5. 16.Pearson TA, et al. (2003) Markers of inflammation and cardiovascular disease: a statement from the CDC and the AHA. Circulation 107(3):499-511. https://pubmed.ncbi.nlm.nih.gov/12551878/
  6. 17.Grice EA, et al. (2009) Topographical and temporal diversity of the human skin microbiome. Science 324(5931):1190-1192. https://pubmed.ncbi.nlm.nih.gov/19478181/
  7. 18.Schoch CL, et al. (2012) Nuclear ribosomal internal transcribed spacer (ITS) region as a universal DNA barcode marker for Fungi. PNAS 109(16):6241-6246. https://pubmed.ncbi.nlm.nih.gov/22454494/
  8. 19.Human Microbiome Project Consortium. (2012) Structure, function and diversity of the healthy human microbiome. Nature 486(7402):207-214. https://pubmed.ncbi.nlm.nih.gov/22699609/

References back the sensor methods, clinical instruments, and lab methods, not SenseMe's proprietary scoring engine. SenseMe uses each instrument under its original published methodology.

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